Because of few sufficiently comprehensive studies, peripheral nerve degeneration has not been universally accepted as a cause of the sensory disturbances in Minamata patients. These patients also frequently presented with signs of sensory neuropathy affecting the distal extremities. Kyodo News. eCollection 2019 Sep 10.Yuan B, Wang DX, Zhu LN, Lan YL, Cheng M, Zhang LM, Chu JQ, Li XZ, Kong DM.Chem Sci. Light and electron microscopic studies.Long-Wavelength Fluorescent Chemosensors for Hg.The MARCKS protein amount is differently regulated by calpain during toxic effects of methylmercury between SH-SY5Y and EA.hy926 cells.Effects of methylmercury on spinal cord afferents and efferents-A review.Neuroimmunological Implications of AQP4 in Astrocytes.
The Daily Yomiuri.
The cats were making strange movements, and falling into the sea. 2016 Aug 10;17(8):1306. doi: 10.3390/ijms17081306. MORIKAWA~ H. ~[ATSUMOTO and Y. SHIRAISHI With 17 Figures in the Text (Received December 20, 1961) Late in 1953, a mysterious illness began to affect the inhabitants, especially fishermen and their families, around Minamata bay situated near the coast in southwestern Kyushu in Japan. Minamata disease. 1950s It started in mid 1950s, in Minamata, Japan, when people began to notice strange occurrences in cats. COVID-19 is an emerging, rapidly evolving situation.The outbreak of methylmercury poisoning in the geographic areas around Minamata Bay, Kumamoto, Japan in the 1950s has become known as Minamata disease. 1997 Nov-Dec;25(6):614-23. doi: 10.1177/019262339702500612.Experientia.
COVID-19 is an emerging, rapidly evolving situation.This site needs JavaScript to work properly. American University, The School of International Service.
2006;108(1):10-23.Toxicol Pathol. At autopsy the dorsal roots and sural nerve showed endoneurial fibrosis, loss of nerve fibers, and presence of Büngner's bands. A case report and a comparative study. It is not well known but it is important to understand in the light of rising environmental threats. Less severe lesions, believed to be responsible for the motor symptoms of Minamata patients, were often found in the precentral, postcentral, and lateral temporal cortices. The present paper describes both biopsy and autopsy findings of the peripheral nerves in a male fisherman who died at the age of 64 years and showed the characteristic central nervous system lesions of Minamata disease at autopsy.
1967 Oct 25;41(10):903-13.Miyagawa T, Deshimaru M, Inoue T, Teraoka A, Hotta N.Seishin Shinkeigaku Zasshi. Minamata disease is a neurological problem caused by methyl mercury poisoning. Tanaka, F. Negligence clear in Minamata case. A case report and a comparative study. With special reference to its pathogenesis.The effects of methyl mercury on morphological and histochemical properties of human and rat spinal cord and cerebellum in tissue culture.The ataxic (cerebellar) form of Creutzfeldt-Jakob disease. The spinal cord showed Wallerian degeneration of the fasciculus gracilis (Goll's tract) with relative preservation of neurons in sensory ganglia. Based on earlier reports and extensive pathological studies on autopsied cases at the Kumamoto University School of Medicine, destructive lesions in … An autopsy case of minamata disease (methylmercury poisoning)--pathological viewpoints of peripheral nerves Toxicol Pathol.
Unrecognized Minamata disease patients to sue government.
The epidemiological study was very well-conducted in a timely manner and the article was available as early as January 1957 in an academic journal … These findings support the contention that there is peripheral nerve degeneration in Minamata patients due to toxic injury from methylmercury.This site needs JavaScript to work properly. [Electron microscopic studies on the rat cerebellum in experimental inorganic mercury poisoning].
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